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Training Desaturation Patterns

LiveO2 fre­quent­ly pro­vokes blood oxy­gena­tion lev­els to devi­ate from what seems nor­mal. These pat­terns often reflect issues with phys­i­ol­o­gy that improve aware­ness of what is hap­pen­ing in the body.

LiveO2 has observed thou­sands of users dur­ing oxy­gen train­ing. Our expe­ri­ence has caused us to expand the expla­na­tions of blood sat­u­ra­tion pat­terns well beyond con­ven­tion­al mod­els. Here are a few discoveries:

  • Oxy­gen in the blood varies wide­ly in dif­fer­ent peo­ple — espe­cial­ly dur­ing exertion
  • Low oxy­gen in the blood often indi­cates high tis­sue con­sump­tion dur­ing first/early ses­sions because tis­sues drain oxy­gen from the blood when the body has a rich sur­plus to repay debt;
  • Low oxy­gen read­ings in the blood are not “dan­ger­ous” as long as the tis­sue has enough oxygen

About Pulse Oximeters

A pulse oxime­ter is a device that shines a light through the fin­ger, and detects pulse rate by “watch­ing” the blood flow tide usu­al­ly in a finger. 

They require an unin­ter­rupt­ed blood flow path from the heart. When users grab bike han­dles their grip often pinch­es the artery that feeds the fin­ger pre­vent­ing the oxime­ter from detect­ing the pulse. 

A pulse oxime­ter shines a red light through the fin­ger to mea­sure the red­ness of the blood. The red­ness of the blood indi­cates the sum of oxy­gen and car­bon monox­ide attached to hemo­glo­bin. Blue­ness reflects the absence of oxy­gen or deoxy­he­mo­glo­bin.

Carbon Monoxide

Pulse oxime­ters can­not dif­fer­en­ti­ate hemo­glo­bin occu­pied with oxy­gen from hemo­glo­bin occu­pied with car­bon monox­ide. Car­bon monox­ide expo­sure caus­es pulse oxime­ters present inac­cu­rate high readings. 

See Also

Abnormal Pulse Oximetry Readings

When a user does not exhib­it nor­mal sat­u­ra­tion then there is one, or more, phys­i­o­log­i­cal pre­vent­ing the the oxy­gen sen­sor from show­ing nor­mal readings. 

Users that have dif­fi­cul­ty get­ting the pulse oxime­ter to pro­vide a read­ing — often have poor circulation.

Pulse Oxime­ters DO NOT mea­sure oxy­gen being deliv­ered to tis­sue — only the amount of oxy­gen that could be deliv­ered when the vas­cu­lar sys­tem works properly. 

See Also

Overview

Some rea­sons why appar­ent blood oxy­gen varies wide­ly in dif­fer­ent peo­ple. There are mul­ti­ple sce­nar­ios based on over­looked but well known observations.

Carbon Monoxide internal or external

Car­bon Monox­ide makes blood red — just like oxy­gen. Pulse oxime­ters PO2 tell is the sum of [Car­bon Monox­ide + O2] in the blood.  Med­ical ref­er­ences pre­sume that oxy­gen in the blood is reli­ably deliv­ered to the tis­sues. This is false for a vari­ety of rea­sons, but usu­al­ly do to an unrec­og­nized inabil­i­ty of the vas­cu­lar sys­tem to deliv­er blood, thus oxy­gen to tissues.

Pres­ence of car­bon monox­ide in the blood caus­es pulse oxime­ters to indi­cate inac­cu­rate high lev­els of oxy­gen. Adhe­sion of Car­bon Monox­ide to hemo­glo­bin is much high­er than oxygen.

In sum­ma­ry — an indi­vid­ual with car­bon monox­ide expo­sure will look like they resist desat­u­ra­tion because it is much hard­er for the body to release car­bon monoxide.

Car­bon Monox­ide may be:

  1. Absorbed from the envi­ron­men­tal contamination;
  2. Be cre­at­ed inter­nal­ly because of dis­ease.

Indi­vid­u­als with car­bon monox­ide attached to hemo­glo­bin only desat­u­rate after train­ing enables the body to remove car­bon monox­ide from the lungs.

Inhibited Oxygen Delivery

There are sev­er­al rea­sons why blood can­not reach and pass through capillaries:

  • Arte­r­i­al dys­func­tion, flac­cid­i­ty, where arter­ies lack the abil­i­ty to reg­u­late blood rout­ing, nor­mal­ly presents as hypoten­sion, low blood pres­sure, due to vas­cu­lar con­stric­tion, car­diac insufficiency
  • Inflam­ma­tion in cap­il­lar­ies pre­vents blood under ade­quate pres­sure to pass through capillaries.
  • Blood agglu­ti­na­tion or aggre­ga­tion where blood cells stick togeth­er so the vis­cos­i­ty of blood inhibits flow;
  • Dehy­dra­tion where the blood plas­ma mass is insuf­fi­cient to main­tain phys­i­cal sep­a­ra­tion of blood cells;
  • Vas­cu­lar con­stric­tion where arter­ies that inhibits blood deliv­ery to tis­sue often with hyper­ten­sion, due to sym­pa­thet­ic stress responses;
  • Insuf­fi­cient car­diac out­put to push blood through capillaries;
  • Expo­sure to Car­bon Monox­ide from envi­ron­men­tal or inter­nal pro­duc­tion from dys­func­tion­al metab­o­lism (shows erro­neous lev­el of blood saturation);
  • Car­bon Monox­ide expo­sure — car­bon monox­ide binds more strong­ly to hemo­glo­bin and obstructs oxy­gen absorp­tion in the lungs.

Desaturation & Resaturation Definitions

Desat­u­ra­tion: — is when the blood sat­u­ra­tion read­ing decreas­es as the amount of oxy­gen or car­bon monox­ide attached to hemo­glo­bin decreases; 

Resat­u­ra­tion: when the blood sat­u­ra­tion read­ing increas­es because of an increase in oxy­gen bound to the hemoglobin.

Saturation and Oxygen Debt

Med­ical texts presents stage a con­fus­ing view of blood sat­u­ra­tion for sev­er­al reasons:

  1. False Highs occur when car­bon monox­ide inter­feres with accu­rate read­ings, and that car­bon monox­ide is a result of dis­ease and inflammation;
  2. False Lows occur when tis­sue con­sump­tion spikes due to high lev­els of tis­sue oxy­gen consumption;
  3. Sym­me­try Assump­tion: Vary­ing read­ings occur when tis­sue on one side of the body uses more oxy­gen on the oth­er due to asym­met­ric oxy­gen consumption;
  4. Oxy­gen Debt is an accu­mu­lat­ed lack of an ener­gy that occurs when a tis­sue is deprived of oxy­gen due lack of oxy­gen delivery;
  5. Oxy­gen Debt Pay­back occurs when oxy­gen deliv­ery is restored to a tis­sue and the tis­sue con­sumes a large amount of oxy­gen often caus­ing sub­stan­tial desaturation.

Asymmetric Pulse Oximetry

Pulse oxime­try is often asym­met­ric. This means that pulse oxime­ters on the left and right-hand show sub­stan­tial­ly dif­fer­ent num­bers. This indi­cates that blood in dif­fer­ent flow cir­cuits, deliv­ered to dif­fer­ent parts of the body, have dif­fer­ent amounts of oxygen.

Many false­ly assume that pulse oxime­try is sym­met­ric. First time LiveO2 users will usu­al­ly observe dif­fer­ent PO2 read­ings in the left and right side. This asym­me­try indi­cates that one side of the body us using more oxy­gen than the other.

  • O2 starts out normal
  • There is usu­al­ly an ini­tial drop-low­er in PO2 on the left or right side
  • This side usu­al­ly cor­re­sponds to a local­ized injury, dis­ease or trauma
  • The ini­tial drop normalizes
  • 2nd drop on the right side as the liv­er func­tion ramps up
  • Nor­mal­iza­tion

Local Oxygen Deficiency causes Asymmetric Oximetry

This pat­tern occurs when there is a local­ized deferred demand as a result of deferred oxy­gen deficit.

  • Stage 0: Injury, dis­ease or stress com­pro­mis­es oxy­gen deliv­ery to a local tissue
  • Stage 1: Nor­mal res­pi­ra­tion does not resolve the deficit;
  • Stage 2: Increased oxy­gen sup­ply ris­es to enable res­o­lu­tion of the deficit;
  • Stage 3: Pulse oxime­try read­ing on the cor­re­spond­ing side drops to reflect ele­vat­ed oxy­gen con­sump­tion on that side;
  • Stage 4: Pulse oxime­try nor­mal­izes (typ­i­cal) on both sides;
  • Stage 5: Pulse oxime­try read­ing on the right side drops as the liv­er kicks on
  • Stage 6: Pulse oxime­try read­ing equal on both sides
  • Nor­mal: Asym­me­try does not occur unless there is a stress pattern. 

The first stage is usu­al­ly stressed, injured or dis­eased con­sumes large amounts of oxy­gen which tem­porar­i­ly reduces oxy­gen read­ings on one side of the body. 

Asymmetric Desaturation Pattern Causes

The fol­low­ing expla­na­tions attempt to explain fre­quent obser­va­tions that make up the pat­terns above. Most pre­sen­ta­tions, above, tend to a result of com­bi­na­tions of the fol­low­ing pat­tern causes.

  • Sys­temic Detox caus­es liv­er to switch on result­ing in right side dip
  • Stressed tis­sue con­sumes large vol­ume of oxygen
  • Acti­va­tion of organs with blood sources fed by the same arte­r­i­al feed.

Symmetric O2 Oximetry

When oxime­try varies the same on both sides then it indi­cates a whole-body local­ized effect. This is a typ­i­cal scenario.

  • Stage 0: Cells lose abil­i­ty to absorb oxy­gen for unknown reason
  • Stage 1: Nor­mal PO2 at rest
  • Stage 2: Increased oxy­gen avail­able at rest
  • Stage 3: Body sens­es increased oxy­gen available
  • Stage 4: Cells hog as much oxy­gen as they can
  • Stage 5: Deficit even­tu­al­ly satisfied
  • Stage 6: Oxy­gen lev­el goes to 99% once deficit satisfied 
  • Stage 7: Body turns of “oxy­gen con­ser­va­tion” and desat­u­ra­tion response normalizes

When you watch some­one — this is a gen­er­al guide to what may be hap­pen­ing inside. This is by no means a com­pre­hen­sive list but here are the caus­es which we feel that con­tribute to var­i­ous pre­sen­ta­tions arranged in esti­mat­ed rate of observation.

Pos­si­ble caus­es of sym­met­ric desat­u­ra­tion pattern:

  1. Cap­il­lary Endothe­lial Inflammation
  2. Wide­spread Hypox­ic Tis­sue from reduced blood flow
  3. Cel­lu­lar clear­ance in stress-relat­ed hor­mones as a Drop and dwell at SPO2 98% dur­ing late phas­es of aggres­sive train­ing — accom­pa­nied by minor shak­ing for 10 – 50 min­utes fol­low­ing session.
  4. Local­ized Hypox­ic Tis­sue from local trau­ma caus­ing asym­met­ric PO2 readings
  5. Release of sys­temic tox­ins caus­ing liv­er chal­lenge which reduces PO2 on right side cor­re­sponds to breath odor and detox flows
  6. Res­pi­ra­to­ry Injury to the lung
  7. Tem­po­rary injury to blood that caus­es agglu­ti­na­tion which caus­es durable sys­temic injury to vas­cu­lar endothelium 
    1. Mim­ics Ischemic Event
    2. Heart Attack in the heart
    3. Stroke in the brain
    4. Durable com­pro­mise in organ func­tion for any oth­er organ
  8. Lac­tic Acid Clear­ance in heav­i­ly mus­cu­lar indi­vid­u­als releas­es and caus­es gall blad­der sen­sa­tions often fol­lowed by bow­el clear­ance (foot­ball players);
  9. Ane­mia result­ing in loss in func­tion­al blood vol­ume for indi­vid­u­als who can­not tol­er­ate exercise

Understanding Healthy Patterns

Healthy desat­u­ra­tion resat­u­ra­tion responses

  • Test‑1 Desat­u­ra­tion Test: When a user switch­es to ‑O2 — Oxy­gen Sat­u­ra­tion decreas­es as a func­tion of exer­tion lev­el and hypox­ic air oxy­gen concentration;
  • Test 2 Resat­u­ra­tion Test: When a user switch­es to +O2 after desat­u­ra­tion below 92%- Oxy­gen Sat­u­ra­tion dips 2 – 5 % low­er then rebounds 99% with­in 10 breaths (often 2);

Any obser­va­tion which does not match these pat­terns indi­cates the user has com­pro­mised tis­sue oxy­gen delivery. 

Slug­gish desat­u­ra­tion indi­cates oxy­gen deliv­ery from the blood is inhibited.

Pro­longed low oxime­try after switch­ing to oxy­gen means that the body is repay­ing oxy­gen debt. 

Do not try more chal­leng­ing pro­to­cols until your resat­u­ra­tion pat­tern is nor­mal. Repeat the chal­lenge and recov­ery until resat­u­ra­tion is rapid.

Desaturation Challenge Testing

  1. Exer­tion on ‑O2 air hypox­ic results in a decrease of PO2 read­ing to a min­i­mum which is dri­ven by exer­tion lev­el and oxy­gen con­cen­tra­tion in air;
  2. Switch to oxy­gen results in a fur­ther decrease in oxy­gen by 2 – 5 points for about 3 – 9 sec­onds, as oxy­gen debt accu­mu­lat­ed dur­ing hypox­ic chal­lenge is repaid;
  3. Rapid rebound to 99% in 15 sec­onds or less after switch to oxygen;
  4. Advanced users dwell longer on low oxy­gen air to dri­ve sat­u­ra­tion lev­els low­er as their train­ing advances;
  5. Advance users mea­sure how many breaths of oxy­gen is nec­es­sary to enable PO2 to return to 99%, opti­mal is only 2 breaths.

Anaerobic Lift Pattern

Advanced users can acti­vate anaer­o­bic metab­o­lism espe­cial­ly with LiveO2 Extreme.

This pat­tern is observed on low-oxy­gen Hypox­ic, as blood sat­u­ra­tion decreas­es to a min­i­mum then increas­es 2 – 5 points while remain­ing on hypox­ic air while main­tain­ing mod­er­ate exertion.

The pulse oxime­try increase while remain­ing on low oxy­gen means the body has switched to pri­mar­i­ly anaer­o­bic ener­gy pro­duc­tion. The body uses most­ly glu­cose, and is prone to gen­er­ate lac­tic acid, when a sprint is acti­vat­ed from this mode. 

This tech­nique is enables mod­u­la­tion of EPO and HGH Human Growth Hor­mone for Anti-Aging and endurance enhance­ment protocols.

Do not attempt these pro­to­cols until you are expe­ri­enced with LiveO2

Unhealthy Patterns

Desat­u­ra­tion indi­cates when oxy­gen is deliv­ered to tis­sue. The abil­i­ty to desat­u­rate is an indi­ca­tor of func­tion­al deliv­ery, health, con­firm­ing that the body is able to deliv­er oxy­gen to cells.

Fail­ure to desat­u­rate indi­cates that the oxy­gen is stuck on hemo­glo­bin and that cells are func­tion­al­ly deprived of oxygen.

Restor­ing the oxy­gen reserve in hemo­glo­bin occurs very quick­ly, usu­al­ly with­in 10 sec­onds. Resat­u­ra­tion that takes longer than this, even in advanced pro­to­cols, usu­al­ly indi­cates pay­ment of oxy­gen debt, thus recov­ery. (This is always good.)

Recovery Patterns

Initial Dipper Pattern On Oxygen

This is a recov­ery pat­tern occurs when a user repays oxy­gen debt when using oxy­gen. This pat­tern indi­cates the a phys­i­o­log­i­cal­ly impor­tant per­cent­age of the user’s body has been oxy­gen deprived and that it is vul­ner­a­ble to dis­ease processes.

The dip indi­cates oxy­gen debt repay­ment. It is often com­mon for the user or observers to “smell” volatile sub­stances in the users breath or in body odor:

  • DEET — insect repellent
  • Per­fume or deodorant
  • Mold
  • Yeast
  • Chem­i­cals includ­ing paint or solvents.

Users who expe­ri­ence this often small odors from air that is cap­tured and then inhaled through the mask. They sense this more evi­dent on low oxy­gen ‑O2. Clear­ance of smells indi­cates suc­cess­ful detox­i­fi­ca­tion as the ses­sion progresses.

This pat­tern indi­cates res­o­lu­tion of recov­er­able chron­ic oxy­gen defi­cien­cy usu­al­ly to tis­sues that have been in pro­longed oxy­gen-defi­cient status.

High oxy­gen con­sump­tion by oxy­gen-deprived cells caus­es short-term ele­vat­ed con­sump­tion observ­able as low pulse oxime­try read­ing all while on high oxy­gen +O2. The degree and dura­tion of the low oxy­gen read­ing indi­cate the amount of oxy­gen used by tis­sues dur­ing recovery. 

Pro­to­col guid­ance — main­tain mod­er­ate to light exer­tion and remain on oxy­gen until pulse oximeter(s) show at least 98%. Do not sprint or use hypoxia.

The 99er Pattern

This pat­tern is com­mon with ill­ness or fatigue. It occurs when the users body can­not resists desat­u­ra­tion, even when on low oxy­gen. It occurs when there is a phys­i­o­log­i­cal obsta­cle pre­vent­ing blood from deliv­er­ing oxy­gen to tissue: 

Cause‑1: — Capillary Shunting

The endothe­lial inflam­ma­tion reduces below the pass­able diam­e­ter of a red blood cell (RBC). When this occurs, only plas­ma can flow through the cap­il­lar­ies, lim­it­ing ener­gy pro­duc­tion to anaer­o­bic fueled by glu­cose absent oxygen.

The unnat­u­ral­ly high PO2 usu­al­ly occurs when blood can­not reach tis­sues due to endothe­lial cap­il­lary inflam­ma­tion. These nor­mal­ly expe­ri­ence fatigue because cells are in an oxy­gen deprived sta­tus because blood shunts around cap­il­lar­ies. See Cap­il­lary Shunting

The reduced cap­il­lary cross sec­tion caus­es RBCs to go around nar­rowed cap­il­lar­ies. RBCs that don’t pass through cap­il­lar­ies do not release oxy­gen much like a vehi­cle that can­not release a pay­load — it just remains full. This shows up as an unnat­u­ral­ly high start­ing PO2 and a ten­den­cy NOT to desat­u­rate dur­ing hypox­ic exer­tion challenge.

This pat­tern has been observed:

  • Fibromyal­gia / Chron­ic Fatigue
  • Can­cer patients after chemotherapy

For com­par­i­son a start­ing sat­u­ra­tion lev­el of about 97%, with
rapid desat­u­ra­tion to 87%, is nor­mal (sea level).

This pat­tern con­tra­dicts the typ­i­cal med­ical con­clu­sion that a high
hemo­glo­bin sat­u­ra­tion indi­cates good tis­sue oxy­gena­tion. The med­ical
inter­pre­ta­tion pre­sumes, usu­al­ly incor­rect­ly, that oxy­gen can always
move from the RBC to tis­sue. By the time this sat­u­ra­tion pat­tern,
99 – 100%, occurs when the person’s body has a large per­cent­age of
under-oxy­genat­ed tissue.

The sever­i­ty of sys­temic hypox­ia is indi­cat­ed by how long it takes
them to re-sat­u­rate after the inflam­ma­tion is reversed. On the pulse
oxime­ter, how many min­utes does it take them to sat­u­rate to 99% after
they resat­u­ra­tion dip? 

The longer the time, the greater the accu­mu­lat­ed
oxy­gen tis­sue debt.

The degree of sys­temic hypox­ia is indi­cat­ed by how long it takes the
per­son to re-sat­u­rate after­ward (the amount of time the per­son spends
on oxy­gen with a low oxy­gen level).

The prob­lem is that the oxy­gen bound to hemo­glo­bin can­not dis­so­ci­ate
because it nev­er pass­es through the cap­il­lar­ies where it can release
oxy­gen. In this case, unnat­u­ral­ly high hemo­glo­bin sat­u­ra­tion means
poor tis­sue oxygenation.

Resolution Pattern

The tell­tale for res­o­lu­tion of this pat­tern is a dra­mat­ic drop in
PO2 late in the ses­sion while on oxy­gen. Here is a mod­el
for what happens:

  1. Cap­il­lary pulse pres­sure reach­es the pen­e­tra­tion thresh­old as
    arte­r­i­al blood pres­sure and hypox­ia-induced vasodi­la­tion deliv­er
    more pres­sure to cap­il­lary bed. This takes effort and some time.
    It does not hap­pen instant­ly, and takes 5 – 10 min­utes of effort.
  2. Endothe­lial cells switch back to nor­mal metab­o­lism and pump-out
    sodi­um and quick­ly shrink back to nor­mal size
  3. Cap­il­lary opens to red blood cell pas­sage and tis­sue reoxy­gena­tion begins
  4. PO2 drops as tis­sues absorb large amount of oxy­gen until
    reper­fu­sion is com­plete, usu­al­ly in 2 – 4 minutes.

Start­ed abnor­mal­ly high 99% PO2 on start and then desat­u­rat­ed to 80% after hypox­ic chal­lenge about 9 min­utes into the ses­sion. User remained at 80% PO2 for 2 min­utes while breath­ing oxy­gen. Nor­mal resat­u­ra­tion time is 5 seconds.

Typical Observation

The tell­tale for res­o­lu­tion of this pat­tern is a dra­mat­ic drop in PO2 late in the ses­sion while on oxy­gen. Here is a mod­el for what happens:

  1. Cap­il­lary pulse pres­sure reach­es the pen­e­tra­tion thresh­old as arte­r­i­al blood pres­sure and hypox­ia-induced vasodi­la­tion deliv­er more pres­sure to the cap­il­lary bed. This takes effort and some time; it does not hap­pen instant­ly, and takes 5 – 10 min­utes of effort.
  2. Endothe­lial cells switch back to nor­mal metab­o­lism and pump out sodi­um and quick­ly shrink back to nor­mal size
  3. Cap­il­lary opens to red blood cell pas­sage and tis­sue re-oxy­gena­tion begins
  4. PO2 drops as tis­sues absorb large amounts of oxy­gen until re-per­fu­sion is com­plete, usu­al­ly in 2 – 4 minutes.

Pathological Hints

This is the typ­i­cal chron­ic-fatigue pat­tern. It usu­al­ly includes per­sis­tent mus­cle touch sen­si­tiv­i­ty from region­al tis­sue aci­do­sis. Over time this con­di­tion can progress to mul­ti­ple local and sys­temic dis­ease states:

  • Hypo­glycemia as under-oxy­genat­ed tis­sues use exces­sive glu­cose. If the liv­er fails to keep up with demand, then blood sug­ar falls to hypo­glycemic lev­els and caus­es sys­temic fatigue.
  • Gall blad­der con­di­tions include dis­com­fort and gall­stones. When the Cori Cycle depletes lac­tic acid reacts with bile in the gall blad­der to pre­cip­i­tate solids which often form gall­stones and cause discomfort.
  • This author sug­gests that tis­sues that retain excess lac­tic acid for a long time become hyper­sen­si­tive as with fibromyal­gia.

See Fatigue Pro­to­col for more information.

Protocol Suggestion

Recurrence

Nor­mal­ly this pat­tern only occurs once dur­ing ear­ly use. Re-per­fu­sion is durable until con­di­tions that caused endothe­lial inflam­ma­tion recur.

LiveO2 Adap­tive Con­trast appears to be a require­ment to pro­voke resat­u­ra­tion. It seems the rea­son for this is that reduced-oxy­gen air cre­ates vasodi­la­tion and increas­es arte­r­i­al pulse pres­sure, which max­i­mizes pulse pres­sure at the cap­il­lary entrance. This re-per­fu­sion effect has not been observed with LiveO2 Stan­dard.

What to Expect

If you expe­ri­enced this pat­tern, you will likely:

  • Feel stronger and have increased endurance
  • Reduced crav­ings for sweets and sim­ple carbohydrates
  • Reduced ten­den­cy for mus­cle soreness
  • Greater strength in major muscles
  • Reduced ten­den­cy for loose stools
  • Improved fat diges­tion from improved bile availability
  • Have an increased res­pi­ra­tion rate at rest

Cause 2 Carbon Monoxide Poisoning

Car­bon Monox­ide binds to hemo­glo­bin about 254x more strong­ly than Oxy­gen. CO caus­es blood to appear unnat­u­ral­ly red trick­ing the Pulse Oxime­ter to reg­is­ter inac­cu­rate­ly high.

Car­bon Monox­ide may be inter­nal­ly pro­duced. So you may exhib­it this absent expo­sure to envi­ron­men­tal car­bon monoxide.

The typ­i­cal­ly occurs from breath­ing in car­bon monox­ide tox­ic gas (CO) in to body large quan­ti­ties. Often with symp­toms headache, dizzi­ness, weak­ness, vom­it­ing, and loss of consciousness.

LiveO2 enables the body to expe­dite release car­bon monox­ide but it requires longer-term train­ing. Con­tact your train­er for advanced support. 

A user with car­bon monox­ide bound to hemo­glo­bin will resist desaturation.

The 100 Pattern

This occurs when a user observes a PO2 of 100%. This pat­tern reflects oxy­gen deliv­ery to cells is severe­ly inhib­it­ed due to one or more prob­lems. This also presents as a stuck at 98% or 99%.

Acute Capillary Shunting

Cap­il­lary struc­tures are based on a cen­tral thor­ough­fare chan­nel that trans­ports blood between the arte­r­i­al and venous networks. 

When the sys­temic net­work of cap­il­lar­ies is sub­stan­tial­ly blocked, blood can­not deliv­er oxy­gen to cor­re­spond­ing tissues. 

When the cap­il­lary sys­tems are sub­stan­tial­ly blocked this fail­ure caus­es blood to return to the heart with oxy­gen instead of car­bon diox­ide. This caus­es the Pulse Oxime­ter to show unusu­al­ly high saturation.

Indi­vid­u­als with this con­di­tion will nor­mal­ly exhibit:

  • Unusu­al­ly large desat­u­ra­tion events dur­ing ear­ly train­ing ses­sions as oxy­gen surges restore cap­il­lary flow and cells con­sume large amounts of oxygen;
  • Asym­met­ric pulse oxime­try read­ings show tis­sues on left vs right open­ing up cor­re­spond­ing to areas with active and stale injuries. Right oxime­try tends to dip more often than left due to high oxy­gen enabling surges of liv­er func­tion. Left side dips tend cor­re­late to oxy­gen restora­tion to injured tissues.

Instruc­tions:

  • Easy does it
  • Con­tin­ue to esca­late training
  • May take 3+ ses­sions to achieve desaturation

Resaturation Patterns

Updated on September 18, 2024
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